Coronary heart disease (CHD) is of degenerative nature which a person will suffer along with age and is a “Silent killer”. The main cause is the habit of consuming food of high fat content. High fat accumulations will facilitate the formation of plaques in the blood vessels (atherosclerosis). When this occurs in the coronary arteries (blood vessels that nourish the heart) then the heart muscle’s need of nutrients is reduced and subsequently leads to Coronary Heart Disease.

To make it easier for you to understand how coronary heart disease evolves, there are 3 main components in the blood vessels that you should know namely, LDL, platelets, and monocytes.

LDL is Low Density Lipoprotein is one type of fat that is slightly bonded to protein. Because it is binding to proteins it automatically becomes free-fat. LDL is of a disruptive nature in the metabolism and can only be digested in small quantities. This LDL is the main cause of blockage of the coronary artery heart.

Coronary heart disease (CHD) is of degenerative nature which a person will suffer along with age and is a “Silent killer”. The main cause is the habit of consuming food of high fat content. High fat accumulations will facilitate the formation of plaques in the blood vessels (atherosclerosis). When this occurs in the coronary arteries (blood vessels that nourish the heart) then the heart muscle’s need of nutrients is reduced and subsequently leads to Coronary Heart Disease.

To make it easier for you to understand how coronary heart disease evolves, there are 3 main components in the blood vessels that you should know namely, LDL, platelets, and monocytes.

LDL is Low Density Lipoprotein is one type of fat that is slightly bonded to protein. Because it is binding to proteins it automatically becomes free-fat. LDL is of a disruptive nature in the metabolism and can only be digested in small quantities. This LDL is the main cause of blockage of the coronary artery heart.

When there is a high LDL accumulation in the blood, the LDL will interact with the right and left walls of the blood vessels which can potentially lead to endothelial cross links being stretched. Under this circumstance LDL is pressed into the lower layers of the blood vessels.

You need to remember that LDL is free fat so it can easily bind to free radicals. There are numerous types of free radicals one of them is Nitric Oxide. Nitric Oxide will bind to LDL and can worsen the quality of the LDL. When the endothelial link is getting bigger, the amount of LDL penetrating into the blood vessels steadily increases.

The body has a protective cell that is a monocyte that will become effective if there is a foreign residue in its place. For example, in the body there is bacteria, the monocytes will be transformed into macrophages and eat the bacteria but, In this case, the monocytes will enter the blood vessels and serve to consume LDL.

Unfortunately, the fat cells have now been bonded to free radicals, so when the monocytes are transformed into macrophages which eat the fat cells, the fat cells then become hard to digest by the macrophages and are ultimately transformed into a cell foam. Accumulation of foam cells and fat cells are the fat deposits under the blood vessels layers.

When there is a high LDL accumulation in the blood, the LDL will interact with the right and left walls of the blood vessels which can potentially lead to endothelial cross links being stretched. Under this circumstance LDL is pressed into the lower layers of the blood vessels.

You need to remember that LDL is free fat so it can easily bind to free radicals. There are numerous types of free radicals one of them is Nitric Oxide. Nitric Oxide will bind to LDL and can worsen the quality of the LDL. When the endothelial link is getting bigger, the amount of LDL penetrating into the blood vessels steadily increases.

The body has a protective cell that is a monocyte that will become effective if there is a foreign residue in its place. For example, in the body there is bacteria, the monocytes will be transformed into macrophages and eat the bacteria but, In this case, the monocytes will enter the blood vessels and serve to consume LDL.

Unfortunately, the fat cells have now been bonded to free radicals, so when the monocytes are transformed into macrophages which eat the fat cells, the fat cells then become hard to digest by the macrophages and are ultimately transformed into a cell foam. Accumulation of foam cells and fat cells are the fat deposits under the blood vessels layers.

Fat deposits can clog the blood vessels or narrow of the veins and cause a drastically decreased blood supply. If this persists it can rip the blood vessel layer and then activate the process of gradual blood clotting. The component that plays the main part in this process are the platelets. Platelet will directly clog the torn blood vessels and form a blood clot. Blood clots are always connected by fibrin threads, fibrin is a protein in the blood that binds to the aggregation cells. Coronary heart disease can also be caused by high blood pressure and blood clotting. It automatically has a blood clot (thrombus) over the blockage of the fat cells.

The fat deposits under the layers of the blood vessels and thrombus will form a block that further narrows down the blood vessel space, and forms a total occlusion. Now the blood flow is sealed off and no more red blood cells can enter into the body due to these blockages. Gradually the heart muscle will now breakdown in fatal regions potentially causing death.

Fat deposits can clog the blood vessels or narrow of the veins and cause a drastically decreased blood supply. If this persists it can rip the blood vessel layer and then activate the process of gradual blood clotting. The component that plays the main part in this process are the platelets. Platelet will directly clog the torn blood vessels and form a blood clot. Blood clots are always connected by fibrin threads, fibrin is a protein in the blood that binds to the aggregation cells. Coronary heart disease can also be caused by high blood pressure and blood clotting. It automatically has a blood clot (thrombus) over the blockage of the fat cells.

The fat deposits under the layers of the blood vessels and thrombus will form a block that further narrows down the blood vessel space, and forms a total occlusion. Now the blood flow is sealed off and no more red blood cells can enter into the body due to these blockages. Gradually the heart muscle will now breakdown in fatal regions potentially causing death.

If you notice, the complexity of the plaque formation process in the coronary arteries makes you think that it takes a comprehensive treatment to deal with coronary heart disease. We also look for various herbal materials and after working with some herbal researchers from other countries finally we finally found 3 herbal formulations that have been tested and clinically proven, among others:

1. The pineapple fruit with ultra high extraction to nano level can be used as an active protease with the most remarkable benefits. We named this product BROMEKINE. Bromekine has been tested in rats adapted to coronary heart disease and after a 6-month observation of remarkable results were recorded. Around 83.72% of rats changed when the incision of the heart's blood vessels was seen to be very clean and free of plaque. Inversely proportional to mice that are not given bromekine, visible blood vessels are full of fat and there is even an occlusion (total congestion).

2. Gambir with a very high extraction level can be used as an antiatherosclerosis to prevent the formation of foam cells due to excessive fat accumulation. We named this product CARIA PLUS. Caria Plus has been tested in 36 mice that were administered with high-fat foods for 2.5 months. Results showed that antiatherosclerosis activity seen in mice given Caria plus by inhibiting the thickening of the aortic wall and the formation of foam cells (p less than 0.05) compared to rats which were not given Caria plus.

3. The type of earthworm Lumbricus rubellus with equally high extraction can act as an angiogenesis and fibrinolytic agent. We named this product LUMBROKINE. The content of has an enzyme in lumbrokine that can stimulate the synthesis of the Growth Factor (EGF and FGF) so that it will form new blood vessels and harmonize the blood circulation throughout all body tissues. Lumbrokine has been under intense research to improve the concentration of EGF up to 10x as well as 5x FGF in patients receiving therapy than control patients. In addition, the fibrinolytic enzyme in Lumbrokine will help destroy the blockage in the blood vessels.

After successfully conducting animal research, we go over to human research. And the result of 100 people showed 71.21% expressed improvement in the clinical symptoms

If you notice, the complexity of the plaque formation process in the coronary arteries makes you think that it takes a comprehensive treatment to deal with coronary heart disease. We also look for various herbal materials and after working with some herbal researchers from other countries finally we finally found 3 herbal formulations that have been tested and clinically proven, among others:

1. The pineapple fruit with ultra high extraction to nano level can be used as an active protease with the most remarkable benefits. We named this product BROMEKINE. Bromekine has been tested in rats adapted to coronary heart disease and after a 6-month observation of remarkable results were recorded. Around 83.72% of rats changed when the incision of the heart's blood vessels was seen to be very clean and free of plaque. Inversely proportional to mice that are not given bromekine, visible blood vessels are full of fat and there is even an occlusion (total congestion).

2. Gambir with a very high extraction level can be used as an antiatherosclerosis to prevent the formation of foam cells due to excessive fat accumulation. We named this product CARIA PLUS. Caria Plus has been tested in 36 mice that were administered with high-fat foods for 2.5 months. Results showed that antiatherosclerosis activity seen in mice given Caria plus by inhibiting the thickening of the aortic wall and the formation of foam cells (p less than 0.05) compared to rats which were not given Caria plus.

3. The type of earthworm Lumbricus rubellus with equally high extraction can act as an angiogenesis and fibrinolytic agent. We named this product LUMBROKINE. The content of has an enzyme in lumbrokine that can stimulate the synthesis of the Growth Factor (EGF and FGF) so that it will form new blood vessels and harmonize the blood circulation throughout all body tissues. Lumbrokine has been under intense research to improve the concentration of EGF up to 10x as well as 5x FGF in patients receiving therapy than control patients. In addition, the fibrinolytic enzyme in Lumbrokine will help destroy the blockage in the blood vessels.

After successfully conducting animal research, we go over to human research. And the result of 100 people showed 71.21% expressed improvement in the clinical symptoms